Stanley Falkow has spent his life studying how bacteria cause human disease. But ask him whose side he’s on, and he’s likely to pause. Or maybe not. Actually, it’s been pretty clear all along - as evidenced by an experiment in graduate school that required him to feed hapless bacteria to a hungry slime mold. “I felt like a traitor,” recalled Falkow, PhD, the Robert W. and Vivian K. Cahill Professor in Cancer Research at the Stanford University School of Medicine.
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A new study has shown that an investigational drug (R207910, currently in clinical trials against multi-drug resistant tuberculosis strains) is quite effective at killing latent bacteria. This revelation suggests that R207910 may lead to improved and shortened treatments for this globally prevalent disease.
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The Centers for Disease Control and Prevention (CDC) launched a national campaign to teach parents how to keep their children safe from skin infections caused by the potentially dangerous bacteria methicillin-resistant Staphylococcus aureus (MRSA). MRSA, a type of staph bacteria that is resistant to certain antibiotics, has been in the news because it can cause severe infections in health care settings, such as hospitals.
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Infectious heart disease is still a major killer in spite of improvements in health care, but the way the disease develops has changed so much since its discovery that nineteenth century doctors would not recognise it, scientists heard at the Society for General Microbiology’s Autumn meeting being held this week at Trinity College, Dublin. Infective endocarditis is a devastating, progressive and frequently fatal heart disease usually caused by bacterial pathogens.
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